Pharm Exam 4 Fillable Worksheet

Complete each section. Click “Generate PDF” to download a summarized copy.

General Tip

Focus on why each drug class works. Understand the pathophysiology behind it. This helps explain side effects and nursing interventions.

1. Anticoagulants / Antiplatelets / Thrombolytics

A. Anticoagulants

Heparin - Mechanism of Action: Heparin - Therapeutic Uses: Heparin - Monitoring/Labs: Heparin - Key Side Effects: Heparin - Nursing Considerations: Heparin - Why does it work? (Link to patho): Warfarin - Mechanism of Action: Warfarin - Therapeutic Uses: Warfarin - Monitoring/Labs: Warfarin - Side Effects: Warfarin - Nursing Considerations: DOACs - Mechanism: DOACs - Advantages/Disadvantages:

B. Antiplatelets

Mechanism of Action: Clinical Uses: Safety Concerns: Why used for arterial clots?

C. Thrombolytics (tPA)

Mechanism: Indications & Time Window: Major Adverse Effect: Nursing Actions:

2. Diuretics & Renal Function

A. Loop Diuretics (Furosemide)

Site of Action: Primary Effects & Uses: Key Electrolyte Considerations: Why is IV push rate important?

B. Thiazide Diuretics (HCTZ)

Mechanism: First-Line Use: Limitations (Renal Function):

C. Potassium-Sparing (Spironolactone)

Mechanism: Why is hyperkalemia a concern? Nursing Education (diet):

D. Osmotic Diuretics (Mannitol)

Primary Indication: Main Adverse Effect:

3. Antihypertensives & RAAS

A. RAAS Inhibitors (ACE, ARBs)

Key Mechanism: Common Adverse Effects: Why hyperkalemia?

B. Beta Blockers (Metoprolol)

Mechanism: Uses in HTN & HF: Nursing (HR checks):

C. Calcium Channel Blockers

Two Subtypes: Primary Effects (BP, HR, conduction): Why avoid grapefruit juice?

4. Cardiac Glycosides

Digoxin - Mechanism: Therapeutic Range: Signs of Toxicity: Interaction with Hypokalemia:

5. Antiarrhythmic Drugs

Class I (IA vs IB vs IC): Class II (Beta Blockers) - Use in Arrhythmias: Class III (Amiodarone) - Key Toxicities: Class IV (Ca²⁺ Blockers) - Primary Action: Other (Adenosine, Atropine) - Indications:

6. Antianginal Medications

Nitrates - Mechanism: Contraindications: SL Administration Steps: BB/CCBs for Angina - Decreasing O2 Demand:

7. Lipid-Lowering Agents

Statins - Mechanism: Major Side Effects: Bile Acid Sequestrants - Tip: Niacin - Flushing & Reduction Strategy: Fibrates - Main Action & Myopathy Risk:

Short Answer / Scenario Practice

Heart Failure Patient

(Furosemide + Digoxin) Critical Labs:

New DVT (Enoxaparin)

Teaching Points (SubQ & Bleeding):

Severe Hypertension (IV Nitroprusside)

Key Monitoring & Reflex Changes:

Angina Attack (SL Nitroglycerin)

Steps & Assessments:

Statin Therapy (Muscle Aches)

Priority Labs/Assessments: Personal Study Notes: